What effect should a bronchodilator have on your asthmatic client?

Increasing airway resistance would worsen bronchoconstriction and exacerbate asthma symptoms, which is the opposite of the therapeutic intent of a bronchodilator.

Bronchodilators act on airway smooth muscle receptors (such as beta-2 adrenergic receptors) to cause relaxation and dilation of the bronchi and bronchioles. This dilation directly reduces airway resistance, allowing air to move more freely into and out of the lungs. In asthma, where bronchoconstriction is a hallmark feature, this effect relieves the obstruction and improves ventilation.

Decreasing blood pressure is not a primary or expected therapeutic effect of bronchodilators; their target is bronchial smooth muscle, not systemic vascular resistance.

Increasing blood pressure is not the intended effect of bronchodilators, and while some agents in this class can have minor cardiovascular side effects, this is not their therapeutic purpose.